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SR-17018 was designed as being a G-protein-biased MOP agonist, but displays numerous pharmacological outcomes which can't be described from the biased signaling hypothesis.

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The biased agonism hypothesis proposes that preferential activation of G-protein over β-arrestin pathways could dissociate analgesia from adverse outcomes:

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The authors declare that every one details supporting the results of this study are introduced inside the paper and its supporting details files. The info that guidance the results of this analyze can be obtained through the authors on fair request.